HGG Community Forums

Can you post the sire and one of the mares as an example? I had thought it was the overo/frame gene that caused the issue. But could be mistaken.

I had not looked at the in game notes yet. You’re right about the Kit genes. It looks like 50% of the time the stud would provide a W1 to off spring and 50% of the time the mare would provide a W2. Both of these present together would be lethal (W1/W2). So Punnet square method would say 25% of the foals created would be lethal.

Only a select few W/W combos aren’t lethal. W15/W15 is viable as is homozygous W20 and W15/W20. There may be more. I scrolled through my pinto horses to see what combos I had.

Here’s a good article the gives an overview (on real horse genetics): https://pmc.ncbi.nlm.nih.gov/articles/PMC10854722/
And a quote from the article: Dominant White alleles result in more extensive white markings and are likely lethal in the homozygous state [25]. W1–W14, W16–W18, W21–28, W30, W31, and W33 have not been observed in the homozygous state, and are predicted to be homozygous lethal.

https://mesa.horsegeneticsgame.com/forum/index.php?p=/discussion/comment/93853#Comment_93853